• Acne
• Digestive Disorders
• Hypoglycaemia
• Osteoporosis

ILLUSTRATIONS OF PRODUCT APPLICATIONS

These examples are taken from the book "An A-Z of Natural Therapy" (A Physicians Desk Reference – part II) written by leading U.S. chiropractor Lee Sargent, D.C. with contributions from U.K. nutritional therapist Michael Sellar, Dip. I.O.N. The recommendations are those of the practitioners, based on their long experience as clinicians.

In addition to the detailed examples shown, the book also gives nutritional advice for patients with the following ailments: Adrenal Dysfunction, Alcoholism, Alkalosis/Acidosis, Allergy, Alzheimer's Disease, Anaemia(s), Anaemia (Sickle Cell), Angina Pectoris, Asthma, Atherosclerosis, Biliary Disorders, Bursitis, Candida Albicans, Cardiac Arrhythmia, Chronic Fatigue Syndrome, Colitis, Congestive Heart Failure, Constipation, Crohn's Disease, Depression, Dermatitis Herpetiformis, Diabetes Mellitus, Dysmenorrhea, Epilepsy, Epstein Barr Virus, Fibrocystic Breast Disease, Gall Bladder Disease, Gingivitis/Receeding Gums, Gout, Hepatitis, Herpes Simplex, Herpes Zoster, "Hyperoestrogenism", Hypertension, Hypochlorhydria, Immunodepression, Infection (Active/Acute), Inflammation, Insomnia, Kidney Dysfunction, Lupus (SLE), Meniere's Syndrome, Menopausal/Post Menopausal, Mitral Valve Prolapse, Multiple Sclerosis, Muscle Cramps, Obesity, Osteoarthritis, Pain, Parasites (see Digestive Disorders) , Parkinson's Disease, Pituitary Dysfunction, Pregnancy, Pre-menstrual Syndrome, Psoriasis, Rheumatism, Rheumatoid Arthritis, Schizophrenia, Sensorlneural Hearing Loss, Sinusitis, Thyroid Dysfunction, Tinnitus, Tiredness, Ulcer (Duodenal and Gastric), Ulcer (Skin), Ulcerative Colitis, Urticaria (Chronic), Vasculitis Purpura, Vtiligo.

For details of all the products named and their availability, contact Enzyme Process (UK).

Both observational and experimental placebo-controlled studies indicate that zinc may be beneficial in acne cases (1-5). One experimental study found that vitamin A may be helpful (6) and its biological utilisation is enhanced with vitamin E supplementation (7).

Other studies have found digestive support is essential for improved assimilation and utilisation of nutrients (8,9). Assessment of pH is helpful in order to understand how and why detoxification mechanisms may be overloaded in these cases.

In females, ovarian support and B6 supplementation may be necessary (10).

CO-ORDINATION SUGGESTIONS: See Acidosis/Alkalosis, Biliary Disorders, Digestive Disorders.

References

1. Pohit et al: Zinc status of Acne Vulgaris patients. J Applied Nutr. 37: 1985
2. Michaelsson, G: Diet and Acne. Nutr. Reviews 39: 1981
3. Michaelsson, G: Brit. J. Dermatology. 97: 1977
4. Hillstrom, L: Brit. J. Dermatology. 97: 1977
5. Michaelsson, G: Brit. J. Dermatology. 96: 1977
6. Kligman, AM et al: Int J Dermatology. 20: 1977
7. Ames, SR: Am. J. Clin Nutr. 22: 1969
8. Barba, A. et al: Dermatologica 165: 1982
9. Rappaport EM: New Engl. J. Med. 252:1955
10. Snider, BL and Dieteman, DF: Arch. Dermatol. 110: 1974

In assessing digestive and nutritional status one must look at the quality of diets Americans habitually consume. Data on food trends since 1900 to the present indicate that the consumption of alkaline and enzyme/nutrient-rich quality foods has declined, while the consumption of foods of acid ash and low enzyme/nutrient quality has increased (1). Digestive status and nutrient/enzyme reserves are also influenced by factors other than eating habits, including cigarette smoking, drug interactions, alcohol consumption and physiological stress (2). Because of these environmental and dietary factors, more and more patients with digestive symptons are in varying states of acidosis. The lowered pH and enzyme responsible for impaired digestion, absorption and resultant decreased nutrient utilisation. Although some researchers have recognised one aspect of digestive incompetence (hypochlorhydria) (3-7), Morter’s pioneering research on pH and digestive function has explained how these problems are related to a decreased pH and lowered enzyme reserve.

CO-ORDINATION SUGGESTIONS: Alkalosis/Acidosis, Constipation, Biliary Disorders.

References

1. Welsh SO, Martson RM: J. Am. Dietic Ass. 81; 1981
2. Reddy BS et al: Adv. Cancer Res. 32; 1980
3. Howitz J et al: Lancet 1;1971
4. Francis HW: Nebraska Med. J. 16; 1931
5. Allison JR, Curtis AC: Arch. Derm. 72; 1955
6. Allison JR: Sourthern Med. J. 38; 1945
7. Bulletin Gen. de Therapeuric abstracted in JAMA 39; 1902

The metabolic rate of energy requirements of each patient are important factors, thus carbohydrate utilisation must be seriously considered. Avoidance of simple sugars and refined carbohydrates helps to reduce the symptons of reactive hypoglycaemia (1), while increasing complex carbohydrates (whole grains, nuts, seeds, vegetables) is helpful in normalising glucose metabolism.

Nutrients such as niacinamide have been associated with glucose metabolism disorders and may be helpful (2). A deficiency of vitamin B6, associated with carbohydrate metabolism, can result in faulty carbohydrate metabolism, while supplementation may be beneficial (3).

Chromium supplementation has also been reported as beneficial in these cases (4). Magnesium supplementation may glucose-induced insulin secretion and maintain glucose at normal levels (5). Although the textbook entitled: "Basics of food allergy", states that 75% of functional hypoglycaemia is caused by allergy, many clinicians are finding that these food allergies are a result of digestive incompetence (6).

CO-ORDINATION SUGGESTIONS: Alkalosis/Acidosis, Alcoholism

References

1. Sanders LR et al: Southern Med. J. 75; 1982
2. Shansky A: Drug & Cosmetic Industry 129(4); 1981
3. Krebs EG, Fisher EH: Vit Horm, 22; 1964
4. Anderson RA et al: Fed. Proc. 43; 1984
5. Magnesium Bulletin vol. 4, no. 2; 1982
6. Breneman JC: Basics of Food Allergy, Springfield, Illinois; 1978

Dietary management is critical in this disorder as studies have documented how a high acid ash diet increased calcium excretion and bone less (1). Too much animal protein has been reported as the major factor in calcium bone loss and this is confirmed by observation studies that report statistics on vegetarians and lacto-vegetarians versus omnivores (2-8). Furthermore, Morter has provided physicians with a rational and reasonable answer to why calcium, utilised in the buffering process, is depleted from the alkaline reserve. His long-term clinical research has shown that the body uses this calcium out of necessity for survival in order to buffer the excess amount of inorganic acids generated by a high acid ash diet.

Caffeine and alcohol, known expansive foods, have also been documented as increasing urinary and faecal calcium loss (9-13).

Folic acid, a necessary coenzyme for the conversion of homocysteine to methionine, has been reported as helpful in some cases (14). A vitamin D deficiency has been associated with osteoporosis (15-17), while supplementation may be beneficial in normalising calcium absorption and effective in treating postmenopausal women with osteoporosis (18-21).

The dietary calcium/phosphorus ratio is another factor that can upset calcium balance (22) as scientists and government statistics confirmed the fact that Americans have virtually doubled their intake of dietary phosphorus over the past 40 years from less than 800 to over 1400mg average intake per day (23).

Magnesium deficiency has also been associated with osteoporosis (24-27). Since a deficiency of hydrochloric acid impairs calcium absorption and increases urinary calcium excretion, the possibility of a gastric hypoacidity condition should be seriously considered (29,30). Morter has presented an excellent explanation of how osteoporosis is related to acidosis and a depletion of the alkaline reserve (31).

CO-ORDINATION SUGGESTIONS: Alkalosis/Acidosis

1. Barzel US: J. Am. Geriatrics Soc. September 1982
2. Spencer H et al: Am. J. Clin. Nutr. 37; 1983
3. Marsh AG: J. Am. Dietetic Assoc. Feb. 1980
4. Sanchez IV et al: Am. J. Roentgenol. 131; 1978
5. Marsh AG et al: Am. J. Clin. Nutr. 37; 1983
6. Ellis FR et al: Am. J. Clin. Nutr. 25; 1972
7. Schuette SA et al: J. Nutr. 110; 1980
8. Allen GH et al: Am. J. Clin. Nutr. 32; 1979
9. Hollingbery PW et al: Am. J. Clin. Nutr. 32; 1979
10. Massey LK, Berg TA: Nutr. Res. 5; 1985
11. Heaney RP, Recker RR: J. Lab & Clin. Med. 99; 1982
12. Bickle DD et al: Ann. Int. Med. 103; 1985
13. Saville PD: J. Bone & Joint Surg. 47A; 1965
14. Brattstrom LE et al: Metabolism 34(11); 1985
15. Harju E et al: Arch, Orthoped. & Trauma Surg. 103(6); 1985
16. Baker MR et al: Brit. Med. J. March 3, 1979.
17. Tsai KS et al: J. Clin. Inves. 3; 1984
18. Riggs BL, Nelson KL: J. Clin. Endocrinol. Metab. 61(3); 1985
19. Nordin BE et al: Am. J. Clin. Nutr. 42(3); 1985
20. Caniggia A et al: Acta Vitaminol. Enzymol. 6; 1985
21. Canigglia A et al: J. Endocrinol. Invest. 7(4); 1984
22. Worthington-Roberts B: Contemporary Developments in Nutrition. St Lewis, 1981
23. Wachman A, Bernstein DS: Lancet, 1; 1968
24. Cohen L, Ketzes R: Israel J. Med. Sci. 17; 1981
25. Mahaffee D et al: Endocrinol. 110; 1982
26. Hahn TJ: Textbook of Endocrinology, New York; 1986
27. Anast CS et al: J. Clin. Endocrinol. Metab. 432, 1976
28. Spencer H, Kramer L: Am. J. Clin. Nutr. 36; 1982
29. Grossman M et al: Gastroenterol. 45; 1963
30. Recker RR: New Engl. J. Med. 313(2); 1985
31. Morter MT: Osteoporosis, Chiropractic Professional, 5/6, 1987

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